6th International Symposium on Intensive Care and Emergency by A. F. Junod (auth.), Dr. J. L. Vincent (eds.)

By A. F. Junod (auth.), Dr. J. L. Vincent (eds.)

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Extra resources for 6th International Symposium on Intensive Care and Emergency Medicine: Brussels, Belgium, April 15–18, 1986

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Since physiologic shunt and hypoxemia result, the hemodynamic consequences of this type of insult might be expected to resemble those of asphyxia, and this is indeed the case. Following near-drowning with freshwater, arterial pressure decreases immediately and then stablizes over several minutes. At the . ~~ I. -m ennd. O. 4 L/mln Fig. 1. Recorded changes in systemic arterial pressure, pulmonary artery pressure, central venous pressure, and cardiac output in a canine model following aspiration of seawater.

They were interested in evaluating the role of sympathetic mechanisms, and aside from the cardiopulmonary dynamics, they also measured lung lymph flow and protein concentration during an increase in intracranial pressure to near mean arterial blood pressure levels. 0 mg/Kg). In the untreated sheep, the increased intracranial pressure resulted in a significant increase in mean arterial blood pressure, systemic vascular resistance, lymph flow, lymph protein concentration, lymph to plasma protein concentration ratio, and lymph clearance and decrease in plasma protein concentration.

Crit Care Med, (in press). 2. Schlag, G, Regele H (1972) Lungenbiopsien bei hypovoliimisch-traumatischem Schock. Med Welt 23:1755-1758 3. Schlag, G, Voigt WH, Schnells G, Glatzl A (1976) Die Ultrastruktur der menschlichen Lunge im Schock. Anaesthesist 25:512-521 4. Schlag, G, Voigt WH, Redl H. Glatzl A (1980) Vergleichende Morphologie des posttraumatischen Lungenversagens. Anaesth Intensive Notfallmed 15:315-339 5. Schlag G (1981) Shock lung. In: DeAngelis J (ed) The high risk patient. Beach International Inc, San Diego, pp 19-29, vol 1 6.

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